Gene | Score gda | Association Type | Type | Original DB | Sentence supporting the association | PMID | PMID Year | ||||
---|---|---|---|---|---|---|---|---|---|---|---|
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0.560 | Biomarker | disease | BEFREE | Stilbenoid pterostilbene complexed with cyclodextrin preserves left ventricular function after myocardial infarction in rats: possible involvement of thiol proteins and modulation of phosphorylated GSK-3β. | 30203709 | 2018 | ||||
|
0.560 | PosttranslationalModification | disease | BEFREE | Exercise preconditioning and low dose copper nanoparticles exhibits cardioprotection through targeting GSK-3β phosphorylation in ischemia/reperfusion induced myocardial infarction. | 29940198 | 2018 | ||||
|
0.560 | PosttranslationalModification | disease | BEFREE | Eplerenone attenuates myocardial infarction in diabetic rats via modulation of the PI3K-Akt pathway and phosphorylation of GSK-3β. | 30323868 | 2018 | ||||
|
0.560 | Biomarker | disease | BEFREE | Moreover, expressions of p-Akt/Akt and p-GSK-3β/GSK-3β in the myocardium of MI group rats were significantly increased by nesfatin-1 administration, suggesting that nesfatin-1, which appears to possess anti-apoptotic and anti-inflammatory properties, may confer protection against ISO-induced MI via an Akt/GSK-3β-dependent mechanism. | 28720397 | 2017 | ||||
|
0.560 | Biomarker | disease | BEFREE | We assessed whether n-butylidenephthalide (BP) primed ADSCs can attenuate arrhythmias by a GSK-3β-dependent pathway after myocardial infarction. | 28130118 | 2017 | ||||
|
0.560 | Biomarker | disease | BEFREE | Deletion of GSK-3β induces a profibrotic myofibroblast phenotype in isolated cardiac fibroblasts, in post-myocardial infarction hearts, and in mouse embryonic fibroblasts deleted for GSK-3β. | 24899689 | 2014 | ||||
|
0.560 | Therapeutic | disease | RGD | Erythropoietin affords additional cardioprotection to preconditioned hearts by enhanced phosphorylation of glycogen synthase kinase-3 beta. | 16565311 | 2006 | ||||
|
0.560 | Biomarker | disease | CTD_human | PARP inhibition prevents postinfarction myocardial remodeling and heart failure via the protein kinase C/glycogen synthase kinase-3beta pathway. | 16716347 | 2006 |