<b>Conclusion:</b> Tand-scFv<sub>Sca-1+GPIIb/IIIa</sub> is a promising candidate to enhance therapeutic cell delivery in order to promote myocardial regeneration and thereby preventing heart failure.
<b>Conclusion:</b> Tand-scFv<sub>Sca-1+GPIIb/IIIa</sub> is a promising candidate to enhance therapeutic cell delivery in order to promote myocardial regeneration and thereby preventing heart failure.
<b>Conclusion:</b> Tand-scFv<sub>Sca-1+GPIIb/IIIa</sub> is a promising candidate to enhance therapeutic cell delivery in order to promote myocardial regeneration and thereby preventing heart failure.
<b>Conclusion:</b> This study demonstrates that CXCR4 pathway contributes to pathogenesis of cardiac fibrosis in dilated cardiomyopathy, and it represents a new potential therapeutic target in heart failure.
<b>Conclusions:</b> Taken together, our study elucidates a novel role for PDE10A in the regulation of pathological cardiac remodeling and development of heart failure.
<i>Conclusion.</i> Ginsenoside Re can improve isoproterenol-induced myocardial fibrosis and heart failure by regulation of the TGF-<i>β</i>1/Smad3 pathway.
(2014) show that upregulation of microRNA-25 impairs calcium handling leading to pump dysfunction and that targeting microRNA-25 using antisense oligonucleotides reverses pump dysfunction and improves survival in mice with heart failure.