Through whole-genome sequencing analysis, we report that a homozygous splice site mutation in coiled-coil domain containing 39 (<i>Ccdc39</i>) is responsible for early postnatal hydrocephalus in the <i>progressive hydrocephal</i><i>us</i> (<i>prh</i>) mouse mutant.
Through whole-genome sequencing analysis, we report that a homozygous splice site mutation in coiled-coil domain containing 39 (<i>Ccdc39</i>) is responsible for early postnatal hydrocephalus in the <i>progressive hydrocephal</i><i>us</i> (<i>prh</i>) mouse mutant.