We aimed to investigate changes in the levels of cytokines (IL-6, TNF-α and IL-10), miRNAs profiles (miR-146 and miR-155) and distribution of different monocyte subsets (CD14++CD16-, CD14++CD16+, CD14+CD16++) in the acute and post-healing phases of AMI.
The expression of unbound FcγRI was significantly decreased in AMI and UA patients and that of unbound FcγRIII was significantly decreased in AMI patients, with no difference in the expression of unbound FcγRII among the four groups.