Chronic Obstructive Airway Disease
|
0.700 |
Biomarker
|
disease |
HPO |
|
|
|
Chronic Obstructive Airway Disease
|
0.700 |
Biomarker
|
disease |
CTD_human |
|
|
|
Anemia, Hemolytic
|
0.400 |
Biomarker
|
disease |
HPO |
|
|
|
Chronic Airflow Obstruction
|
0.300 |
Biomarker
|
disease |
CTD_human |
|
|
|
Heme Oxygenase 1 Deficiency
|
0.300 |
Biomarker
|
disease |
CTD_human |
|
|
|
Hematuria
|
0.100 |
Biomarker
|
phenotype |
HPO |
|
|
|
Hepatomegaly
|
0.100 |
Biomarker
|
phenotype |
HPO |
|
|
|
Proteinuria
|
0.100 |
Biomarker
|
phenotype |
HPO |
|
|
|
Growth delay
|
0.100 |
Biomarker
|
phenotype |
HPO |
|
|
|
Alzheimer's Disease
|
0.400 |
Biomarker
|
disease |
BEFREE |
Using immunoblotting methods, we further showed that HO-1 protein content was increased in neocortical and vascular samples from AD subjects compared with controls.
|
7543935 |
1995 |
Glioblastoma
|
0.060 |
AlteredExpression
|
disease |
BEFREE |
In a human glioblastoma cell line, T98G, treatment with any of three types of NO donors--sodium nitroprusside, 3-morpholinosydnonimine, and S-nitroso-L-glutathione--caused a significant increase in the levels of heme oxygenase-1 mRNA but not in the levels of heme oxygenase-2 and heat-shock protein 70 mRNAs.
|
8764571 |
1996 |
Glioblastoma Multiforme
|
0.060 |
AlteredExpression
|
disease |
BEFREE |
In a human glioblastoma cell line, T98G, treatment with any of three types of NO donors--sodium nitroprusside, 3-morpholinosydnonimine, and S-nitroso-L-glutathione--caused a significant increase in the levels of heme oxygenase-1 mRNA but not in the levels of heme oxygenase-2 and heat-shock protein 70 mRNAs.
|
8764571 |
1996 |
Adult Glioblastoma
|
0.050 |
AlteredExpression
|
disease |
BEFREE |
In a human glioblastoma cell line, T98G, treatment with any of three types of NO donors--sodium nitroprusside, 3-morpholinosydnonimine, and S-nitroso-L-glutathione--caused a significant increase in the levels of heme oxygenase-1 mRNA but not in the levels of heme oxygenase-2 and heat-shock protein 70 mRNAs.
|
8764571 |
1996 |
Childhood Glioblastoma
|
0.050 |
AlteredExpression
|
disease |
BEFREE |
In a human glioblastoma cell line, T98G, treatment with any of three types of NO donors--sodium nitroprusside, 3-morpholinosydnonimine, and S-nitroso-L-glutathione--caused a significant increase in the levels of heme oxygenase-1 mRNA but not in the levels of heme oxygenase-2 and heat-shock protein 70 mRNAs.
|
8764571 |
1996 |
Renal Cell Carcinoma
|
0.040 |
Biomarker
|
disease |
BEFREE |
Quantitative measurement of heme oxygenase-1 in the human renal adenocarcinoma.
|
8913885 |
1996 |
Hypertensive disease
|
0.600 |
AlteredExpression
|
group |
BEFREE |
For comparison, HO-1 levels were assayed in RPE cells from four donor eyes with long-standing hypertension.
|
9134328 |
1997 |
Alzheimer's Disease
|
0.400 |
GeneticVariation
|
disease |
BEFREE |
Here, we show that a (GT)n repeat in the human HO-1 gene promoter region is highly polymorphic, although no particular alleles are associated with AD or PD.
|
9225984 |
1997 |
Parkinson Disease
|
0.400 |
GeneticVariation
|
disease |
BEFREE |
Here, we show that a (GT)n repeat in the human HO-1 gene promoter region is highly polymorphic, although no particular alleles are associated with AD or PD.
|
9225984 |
1997 |
Hemochromatosis
|
0.200 |
Biomarker
|
disease |
MGD |
Heme oxygenase 1 is required for mammalian iron reutilization.
|
9380735 |
1997 |
Disorder of eye
|
0.010 |
AlteredExpression
|
group |
BEFREE |
This was a limited study; additional screening from different donor eyes will be done in order to establish the relationship between RPE, HO-1 expression and eye diseases in which oxidative stress is believed to be a determinant in the pathophysiology.
|
9134328 |
1997 |
Anemia, Hemolytic
|
0.400 |
Biomarker
|
disease |
CTD_human |
Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency.
|
9884342 |
1999 |
Anemia, Hemolytic, Acquired
|
0.300 |
Biomarker
|
disease |
CTD_human |
Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency.
|
9884342 |
1999 |
Anemia, Microangiopathic
|
0.300 |
Biomarker
|
disease |
CTD_human |
Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency.
|
9884342 |
1999 |
Blood Coagulation Disorders
|
0.300 |
Biomarker
|
group |
CTD_human |
Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency.
|
9884342 |
1999 |
Iron Metabolism Disorders
|
0.300 |
Biomarker
|
group |
CTD_human |
Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency.
|
9884342 |
1999 |